Single Protein Can Heal Neurodegenerative Diseases like Parkinson’s, Huntington’s, Alzheimer’s and ALS

A recent scientific advancement has enabled doctors and physicians to heal a number of demoralizing Neurodegenerative Diseases. A team of Researchers from the US’s Gladstone Institutes has developed a key cellular protein which can effectively heal multiple Neurodegenerative maladies like Parkinson’s, Huntington’s, Alzheimer’s and amyotrophic lateral sclerosis (ALS).

All such health issues and anarchies are generated by mischievous proteins in the brain. Such proteins misplaced and built up in neurons, causing wreaking damage and finally carnage the cells of the brains. Till now, no such particular medicine or therapy has been invented which can detect such Neurodegenerative issues at their primitive stage and heal them easily. But the newly developed Protein has triggered the possibility of healing all such Neurodegenerative problems like Parkinson’s, Huntington’s, Alzheimer’s and amyotrophic lateral sclerosis (ALS) easily and effectively.

In the study, the scientists from the Gladstone Institutes in California, made the use of a special protein type, called ‘Nrf2’ and tried restoring the levels of such disease which holds the potential to a regular and healthy range, thereby defeating the death of brain cells. The scientists tested Nrf2 in two models of Parkinson’s malady: cells with transformations in the proteins LRRK2 and alpha-synuclein. By setting off the Nrf2, the analysts triggered multiple “house-cleaning” tools in the cell to take out overload LRRK2 and alpha-synuclein.

In the research paper, the researcher, Steven Finkbeiner from Gladstone Institute mentioned: ‘We took the model of Parkinson’s disease, ALS, and Huntington’s disease to test Nrf2, and the result was quite positive. We found the single protein preventing all these diseases. Based on the scale and the extensiveness of the effect, we are furthering researching for understanding the role of Nrf2 in regulating the cells and protein in a better way”.

The lead author of the study, Gaia Skibinski, a Ph.D. student and a staff research scientist at Gladstone said, “Excess convention of Nrf2 in cellular models of Parkinson’s disease triggered multiple positive effects. It not only defends the cells against the infection but also works with higher and better effectiveness than any other treatment methods found previously.

The complete details of the research were published in the journal ‘Proceedings of the National Academy of Sciences’.